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Dept. of Orthodontics
Ragas Dental College
ORTHODONTICS INCLUDES THE STUDY OF GROWTH AND
DEVELOPMENT OF JAWS AND FACE PARTICULARLY AND
THE BODY GENERALLY , AS INFLUENZING THE POSITION
OF THE TEETH ;THE STUDY OF ACTION AND REACTION
OF INTERNAL AND EXTERNAL INFLUENCES ON THE
DEVELOPMENT , AND THE PREVENTION AND
CORRECTION OF ARRESTED AND PEVERTED
DEVELOPMENT
Classification of Malocclusion
Etiology
ETIOLOGY
Specific Causes of M.O.
Disturbancesinembryologicdevelopment
Skeletalgrowthdisturbances
Muscledysfunction
AcromegalyandHemi-mandibularhypertrophy
Disturbancesofdentaldevelopment
GeneticInfluences
EnvironmentalInfluences
Equilibriumtheoryanddevelopmentofthedentalocclusion
Functionalinfluencesondentofacialdevelopment
EtiologyinContemporaryperspective
Changingviewsofetiologicpossibilities
EtiologyofcrowdingandMalalignment
EtiologyofSkeletalproblems
M.O.isadevelopmentalcondition
Moderatedistortionofnormaldevelopmentandnotbypathologicprocess
Complexinteractionamongmultiplefactorsthatinfluencegrowthand
developmentandoccasionallyasinglespecificcauseisapparent(eg)
Mand.DeficiencysecondarytochildhoodfractureoftheJaws.
M.O.
Specific causes
Disturbancesinembryologicdevelopment
Geneticdisturbancestospecificenvironmentalinsults.
Teratogens Effect
.Aspirin,cigarettesmoking
(hypoxia) Cleftlipandpalate
Dilantin,valium
Aminopterin Anencephaly
CMV Microcephaly,hydrocephaly,
Micropthalmia
Xradiation Microcephaly
Vitamin`DexcessPrematuresutureclosure
Ethylalcohol Mid-facedeficiency
Skeletal growth disturbances
Fetalmouldingandbirthinjuries.
Intra-uterinemoulding-
Pressureagainstthedevelopingface
Armpressedacrosstheface-Severemaxdeficiency
Decreasedamnioticfluidvolume-headtightlyflexed
againstchest-Mandibledeficiency
Extremesmallmandibleusuallyassociatedwithcleftpalate
(e.g.)pierrerobinsyndrome.
Pierrerobinsyndromewithdefectsincartilageformation-
SticklerSyndrome
Forcepsdelivery-MightdamageTMJ-Internalheamorrhage,lossof
tissueandasubsequentunder-developmentofthemandible.
Injurytothemandibleduringatraumaticdeliveryappearstobearare
andunusualcauseoffacialdeformity.
Hencechildrenwithdeformitiesinvolvingmandiblearemorelikelyto
haveacongenitalsyndrome(e.g.)TreacherCollinsandcruzons
syndrome.
Childhood fractures of the jaw
Condylarneckofthemandibleismorevulnerable.
Condylarprocesstendstoregeneratewellafterearlyfractures.
Prognosisisbetter,theearlierthecondylarfracture,becauseofthe
greatergrowthpotential-earlyinlife.
Itleadstoasymmetricgrowth.
ThoughitisapotentialcauseforsevereM.O.itdoesnotmakealarge
contributiontothetotalpoolofpatientswithmalocclusion.
MaxdownwardbyCranialbaseandforwardbyNasalseptum
Mandpulledforwardbysoft-tissuematrix.
Hence,enoughsoft-tissuescarring-Restrictsthegrowth-Asymmetrical
growth
Managementofcondylarfractures-
Surgicalreduction- Scarring
Conservativemanagement-Earlymobilizationofjawsto
Minimizeanyrestrictiononmovement
Oldcondylarfractures
Rheumatoidarthritis
Hemifacialmicrosomia
(congenitalabsenceoftissue).
Hence,condylar#atanearlyagegoodprognosis.
Muscle dysfunction
Itaffectsthejawgrowthbytwoways
1. Pointofmuscleattachment.
2. Importantpartofthetotalsofttissuematrix.
Apartofmusculaturelostdue
1. Unknowncause
2. Birthinjury
3. Motornervedamage
ExcessivemusclecontractionTorticollis-Sternocleido-Mastoid
muscle-Twistingofthehead-Restrictsthegrowth
Decreasedtonicmuscleactivity
1. Musculardystrophy
2. Cerebralpalsy
3. Muscleweaknesssyndromes
4. WillleadtoincreasedAnteriorfacialheight-Distortionoffacial
proportionsandmandform-Excessiveeruptionofpostteeth-Const
maxarchanterioropenbite.
Acromegaly and Hemi-mandibular Hypertrophy
Acromegaly -Anteriorpituitarytumor-increasedgrowthharmone-
Excessivegrowthofmandible-cIIIIM.O.
Tumorisremoved(or)irradiated.
Hemi-mandibular hypertrophy -
Unilateralexcessivemandibulargrowthoccursinpatients
(metabolicallynormal).
Etiologyunknown
Mostcommoningirls15-20years.
Condylarhyperplasia
Treatment
excessivegrowthmaystopspontaneously
removaloftheaffectedcondyle&Reconstruction
Multiplesupernumeraryteeth-(e.g.)cleidocranialdysplasia.
. Ectopic Eruption
Malpositionofthetoothbudwillleadtotheeruptionofthetoothina
wrongplace-EctopicEruption.
(e.g.) MaxIstMolartoomesial
MandIIndpremolarsEruptdistally-Endupinramus
Maxcanine -Longpathwayoferuption
Transposition
Early loss of primary teeth
-Mesialdriftofposteriorteeth-eruptsmesially&occlusally
-Distaldriftoftheanteriorteeth(e.g.)prematurelossofC
1. Activecontractionofthetrans-septalfibresingingiva.
2. Pressuresfromlipsandcheeks.
-Decreaseinthearch-perimeterandarch-lengthleadingto
crowdingandmal-alignment.
-Preventionbyspacemaintainer.
DentalTraumacanleadto
Damagetothepermanenttoothbudsfromaninjurytotheprimaryteeth.
Driftofthepermanentteethaftertheprematurelossoftheprimaryteeth.
Directinjurytopermanentteeth.
Traumatotheprimaryteeth
1.Duringpermanenttoothcrownformation-Enameldefects
2.Duringroofformationofpermanenttooth-Rootformationmaystop-
shortenedroots
- Dilaceration-Distortionofrootformatananglewhichaffects
theeruption
TREATMENT
Traumatically displaced tooth buds in children should be
repositioned as early as possible, so that, the root formation is
continuedwithoutanyobstruction.
Traumatothepermanentteeth
1. Damagetotheroots
2. Alveolusfracture
-Afteranaccidentortraumathetoothusuallycanbemovedtotheoriginal
position easily, because after healing (2-3 weeks) ankylosis may
develop.
Genetic Influences
M.O.couldbeinheritedbytwoways
1.Inheriteddisproportionsbetweenteethsizeandjawsize.
2. Inherited disproportions between size and shape of the U/L
Jaws.
1.Comparingthemono-zygoticanddizygotictwins.
2.Comparingthemother-child,father-childandsibling
pairs.
Lauwerynsetal,40%ofdental&facialvariations
leadingtoM.O.canbeattributedtohereditaryfactors.
Environmentalinfluencesduringgrowthanddevelopmentofface,
jaws and teeth consists largely of pressure and forces related to
physiologicactivity.
Relationshipbetweenanatomicformandphysiologicfunction.
Equilibrium theory
Tooth contacts
mastication very heavy very short
swallowing light very short
4. Major equilibrium influences for the teeth should be the light but
longlastingpressuresfromtongue,lipsandchecksatrest.
6. Themajorequilibriuminfluencesonthejawsshouldbepositional
changesaffectingthefunctionalprocess,includingthecondylarprocess.
Functional influences on Dentofacial development
MASTICATORY FUNCTION
Itcouldaffectintwoways
1. Greater use of the jaws, with higher (or) prolonged biting force
couldincreasethedimensionofthejawsanddentalarches.Lessuse
of the jaws might lead to under-development of dental arches and to
crowdingandirregularteeth.
(e.g) Eskimos with broad dental arches as an adaptation to
extremestress.
Theeffectonarchwidthisnotclear.
Difference in muscle strength - biting force is a etiology for long and
shortfaceproblems?
Itwasconcludedthat,theforceexertedbymasticatorymuscleis
notamajorenvironmentalfactorincontrollingtootheruptionandnotan
etiologicfactorformostpatientswithdeep(or)openbite.
-Sucking Habits
- Tongue thrusting
- Respiratory Pattern
ETIOLOGY IN CONTEMPORARY PERSPECTIVE
1. EdwardAngleandhiscontemporariesstatedthatM.O.wasadisease
ofcivilizationandblameitonimproperfunctionoftheJaws(19th
Century)
2. Classical(Mandelian)geneticsdevelopedrapidlyinthe1stpartofthe
20thCenturystatedthatM.O.isprimarilytheresultofinherited
dentofacialproportions.
3. Inthe1980'stherewasastrongswingbacktowardtheearlierview.
4. In21stCentury-Amorebalancedviewemerged.Itwasconcluded
thattheetiologyofmostorthodonticproblemsaredifficulttoisolate,
becauseseveralinteractingfactorsprobablyplayedarole.
Etiology of crowding and Alignment
-MinorClassIproblems,especiallynon-skeletalcross-bite,oftenare
causedprimarilybyalterationinfunction.Majorproblemsusuallyhave
anadditionalgenetic(or)developmentcomponent.
Malformation(or)MalrelationofJaws.
Inheritedpatterns,defectsinembryologicdevelopment,trauma,and
functionalinfluences.
Mandretrogandprog,Maxdeficiency-inheritanceplaysmajorrole
andtheconditionmadeworsebyenvironmentalefforts.
Altered function:VerticalgrowthproblemsusuallyAnterioropen
bite-Thumbsucking-TongueThrusting-Respiratory
problems.
Openbite-Blacksanddeepbite-whites.
MALOCCLUSION
GeneticComponent
OrInheritance
Developmentalproblems
Alteredfunction
ECTODERMAL DYSPLASIA
MASSETER MUSCLE DYSFUNCTION
ACROMEGALY-ANT PITUITARY TUMOR
ACHONDROPLASIA
EQUILIBRIUM THEORY
TREACHER COLLINS SYNDROME
HEMIFACIAL MICROSOMIA
ADENOID FACIES