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Infarction
with
ST segment
PresentedElevation
by :
Aqilah Salha Bt Ahmad Kasmiri
Supervisor :
dr. Pendrik Tandean,SpPD
PATIENT IDENTITY
Name
No.MR
Age
Gender
Date of admitted
: Mr. H.S
: 436760
: 51 years old
: Male
: 27th July 2010
HISTORY TAKING
Chief complaint: Shortness of breath
History taking:
Felt for the last 2days, which happen in a sudden
especially when patient walked 10m after he woke
up in the morning. Its not influenced by weather. He
always sleep using 2-3 pillow. At night, he usually
woke up due to shortness of breath. Chest pain (+),
felt weighted on his left chest for 5 minutes, pain
penetrates to the back of the body and shoulder.
The pain did not improved by rest or medication.
Sweating (+) , Dyspnea (+), Nausea (-)Vomiting (-),
Epigastric pain(-)Cough(-),Defecation & urination is
normal
RISK FACTOR
Gender
Age
: Male
: 51 years old
Heavy smoker, he had smoked 1-2 box of
cigarettes per day for 30 years.
Hypertension (+) since 3 years ago
Dyslipidemia (+)
Past history of cardiovascular disease(-)
Diabetes Mellitus (-)
PHYSICAL EXAMINATIONS
General Appearance :Mild-illness /normal BW /conscious
Vital Sign :
Blood Pressure
Pulse
Respiratory rate
Temperature
: 160/100 mmHg
: 88 bpm, regular
: 28 tpm ; thoracoabdominal
: 36,8 C (per axilla)
Head Examination :
Eyes : anemia(-), icterus(-), cyanosis(-)
Neck : JVP R+2 cmH20
Thoracic Examination :
Inspection
: Symmetric left and right
Palpation
: No mass, no tenderness
Percussion
: Sonor
Auscultation : Breath Sound : vesicular, Rh -/-, wh +/+
Cardiac Examination :
Inspection
: Ictus Cordis not visible
Palpation
: Ictus Cordis not palpable
Percussion
: Normal heart size increase
Auscultation
: Regular of I/II Heart Sound, murmur (-)
Abdominal Examination :
Inspection
: Flat and following breath movement
Palpation
: Liver and spleen unpalpable
Percussion
: Tympani
Auscultation
: Peristaltic sound (+) , normal effect
Extremities :
Oedema pretibial -/-, feel warm +/+
CHEST X-TRAY
Conclusion:
Cardiomegaly, dilatation et elongation aortae.
Alveolar Edema
ECG INTERPRETATION
Conclusion:
1.Sinus tachycardia (HR: 100x/min)
2.Left axis deviation
3.Right Atrial Enlargement
4.Left Ventricle Hipertrophy
5.Anterior wall Myocardial Infarction
LABORATORY
FINDINGS
ECHOCARDIOGRAPHY
Conclusion:
D/ CAD
WORKING DIAGNOSIS
STEMI anterolateral
TIMI 7/16, KILLIP III
Hypertension Grade
II
Dyslipidemia on
treatment
TREATMENT
Total bed rest
O2 2-4 L/min
IVFD NaCl 0.9% 16 drips/min
Anti-platelet aggregation: Aspilet
80mg (0-1-0)
Clopidogrel 75mg
(1-0-0)
Venodilation:Farsorbid 5mg ,
sublingual
:Farsorbid 10mg (1-1-1)
Anti-trombotic: Lovenox
0.6cc/12hrs/IV
ACE Inhibitor:Captopril 25mg (1/2-0-
Acute Myocardial
Infarction
DEFINITION
Myocardial infarction (MI) is rapid
development
of
myocardial
necrosis caused by a critical
imbalance between the oxygen
supply
and
demand
of
the
myocardium.
It results from plaque rupture with
PATHOPHYSIOLOGY
Occurs when coronary blood
most
occurs
cases, infarction
when
an
RISK FACTORS
Age > 45 years old
Male gender
Smoking
Poorly controlled
hypertension
Hypercholesterolemia /
dyslipidemia
Diabetes mellitus
CLINICAL FEATURES
Chest pain >30 minutes
Feels tight, crushing, and
band like
Location in substernal area
Radiate to left arm, throat,
and jaw
Associated features
including palpitation,
sweating, breathlessness,
TIMI Prognosis in
STEMI
Risk Factor
Score
History of
1/1/1
angina/hipertension/
DM
Systolic BP <100
Killip II-IV
Anterior MI or LBBB
Risk
of Death in
RISK30
FACTORS
days
Total
Score
0
1
2
3
4
5
6
7
8
9-16
0.8%
1.6%
2.2%
4.4%
7.3%
12.4%
16.1%
23.4%
26.8%
35.9%
WHO Diagnostic
criteria
1. Clinical history of
Diagnose
Signs of myocardial ischemia
ECG
ST segmen elevation ?
Yes
Acute Myocardial
Infarction
( Q-wave, non-Q
wave )
No Lab
Biochemical cardiac markers ?
No
Yes
NSTEMI
( No ST-Segment
Elevation
Myocardial
Infarction )
Unstable Angina
ECG
12-lead electrocardiogram showing STsegment elevation (orange) in I, aVL and V1V5 with reciprocal changes (blue) in the
inferior leads, indicative of an anterior wall
myocardial infarction.
MANAGEMENT
COMPLICATION of MI
Based on KILLIP classification:
Classification
Description
KILLIP I
KILLIP II
KILLIP III
KILLIP IV
Cardiogenic shock
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