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MARIJUANA
What is marijuana?
Marijuana is the word used to describe the dried flowers, seeds and
leaves of the Indian hemp plant. On the street, it is called by many other
names,
such
as:astro
turf, bhang, dagga, dope, ganja, grass, hemp, home grown, J, Mar
y Jane, pot, reefer, roach, Texas tea and
weed.
Hashish is a related form of the drug, made from the resins of the Indian
hemp plant. Also called chocolate, hash or shit, it is on average six times
stronger than marijuana.Cannabis describes any of the different drugs
that come from Indian hemp, including marijuana and hashish.
Regardless of the name, this drug is a hallucinogena
substance which distorts how the mind perceives the world you
live in.
The chemical in cannabis that creates this distortion is known as
THC.(Tetrahydrocannabinol). The amount of THC found in any given
batch of marijuana may vary substantially, but overall, the percentage of
THC has increased in recent years.
2. Cognitive Dysfunction
Chronic Cannabis Syndrome, better known by the term
Amotivational Syndrome is cognitive impairment in chronic users
which resulted in a decrease in the ability to plan or get the
purpose of life; then causes the user is placed in a job that
requires only low-level cognition.
Research on humans and animals indicate that the use of
cannabis at an early age long impact on cognition and increase
the likelihood neuropsikis disorders (Schneider, 2008). Wilson et
al. (2000) compared the long-term marijuana users who start use
a marijuna at the age of 17 years and below 17 years and above.
In marijuana users which began at the age of 17 years and below
shows the low percentage of gray matter (neurons and dendrites,
where the thought process occurs) and the high percentage of
white matter (myelinated axons) compared with whole brain
volume.
3. Mental illness
Many studies show that there is a correlation between the use of cannabis with
high risk of psychosis and some studies have found a link between the dose
response of psychosis (Fergusson, Poulton, Smith, & Boden, 2006; Zammit,
Allebeck, Andreasson, Lundberg, & Lewis, 2002). after browsing through the
available evidence, of the many results of published research can be concluded
that cannabis use may increase the risk of psychosis in young people, but it
has a significant effect on those who have a predisposition psychosis.
4. Cancer
Research in this area manunjukan many contradictions. Sidney, Quesenberry,
Friedman, & Tekawa (1997) found that smoking marijuana is not at all
associated with an increased risk of cancer. Meanwhile, in different places,
Fligiel (1997), found that histological and molecular changes in the bronchial
epithelium similar between smoking cannabis with tobacco smoking. The
conclusion that can be drawn while, there have not been enough studies to
provide an evaluation that is strong enough to suggest that smoking marijuana
associated with cancer risk.
5. Reproduction
One study found that marijuana use is associated with decreased
testosterone levels, sperm count and sperm motility in chronic users /
intensive cannabis (Kolodny, Masters, Kolodner, & Toro, 1974). One result,
until recently known as a substance that marijuana can cause male
infertility. In fact, no study has ever seen a link between the use of
cannabis and male fertility.
6. Respiratory Effects
Gong, Fligiel, Tashkin, and Barber (1987) showed that smoking marijuana
rolled 3-4 in the same day with a 20-rolled tobacco smoked in a day and
cause cough, wheeze, and sputum production. Recent research has failed
to find a link between long-term marijuana pengggunaan with FEV1 / FVC
(forced expiratory volume in the first secondsetelah take a deep breath),
diffusion capacity, or hyperactive breathing as occurs in tobacco smokers
(Tetrault, Crothers, & Moore, 2007).
Cancer
Alzheimer
Bipolar
Parkinson
Hepatitis A and C
Influenza
7. Ebola
8. HIV
9. Lhympoma
1.
2.
3.
4.
5.
6.
Small blood clots in the blood vessels of the socalled DIC [Disseminated Intravascular
Coagulation] cause coagulopathy [blood clotting
disorders or excessive bleeding] where blood
would not clot simultaneously with DIC [bleeding
and clotting occurs at the same time]. Symptoms
of shock against toxins [Toxic Shock Syndrome]
occurs when cytokines are released causing blood
vessels to dilate in such a way.Marijuana
compounds proven to reduce and prevent against
toxic shock and DIC (2)
Ebola virus also attacks the attachment of the cells caused by killer
cells immune to release VEGF [vascular endothelial growth factor:
signaling proteins produced by cells that menstimulasivasculogenesis
and angiogenesis. It is part of a system that restores oxygen supply to
tissues when blood circulation is inadequate] which resulted in the
destruction of the tight relationship between the cells and lead to
leaking of fluid between cells until bleeding.
Cannabis compounds can inhibit VEGF processes that occur in the
relationship between cells. Obstructing VEGF by cannabis compounds
may prevent bleeding such cells.Cannabis compounds can inhibit
VEGF and inhibit glioma brain tumors [type of tumor that begins in the
brain or spinal cord]. Very reasonable to estimate that cannabis
compounds that inhibit VEGF and other cytokines when Ebola virus
attack will help the survival of this deadly virus. Stopping the release
of cytokines aka a major feature of the treatment of this deadly
disease.
References:
1) Antibacterial Cannabinoids from Cannabis sativa: A Structure - Activity Study
Antibacterial Cannabinoids from Cannabis sativa: A Structure - Activity Study;
Giovanni Appendino et al. The School of Pharmacy, University of London
2) Protection Against Septic Shock and Suppression of Tumor Necrosis Factor
and Nitric Oxide Production by Dexanabinol (HU-211), a cannabinoid
Nonpsychotropic Ruth Gallily1, Aviva Yamin1, Departments of Immunology, The
Hebrew University, Faculty of Medicine, Jerusalem, Rehovot, Israel .
3) cannabinoid derivatives US patent 20,070,179,135 A1
4) Treatment of HIV and diseases of immune dysregulation US 20,080,108,647
A1
5) Curr Pharm December 2006; 12 (24): 3135-46. Cannabinoids, immune system
and cytokine network. Massi PVaccani AParolaro D, University of Insubria, Via A.
da Giussano 10, 21052 Busto Arsizio (VA), Italy
6) Cancer Res August 15, 2004 64; 5617 Cannabinoids inhibit the Vascular
Endothelial Growth Factor Pathway in Gliomas Cristina Blzquez HYPERLINK
"http://cancerres.aacrjournals.org/content/64/16/5617.full"1,
7) How Cannabis Might Keep Coronary Stents Open Longer,
www.cbds.com/.../how-cannabis-might-keep-coronary-stents-open-longer, June
10, 2014 David Allen MD