2. SUBACUTE PHASE - DURING ADMISSION - WOUND, INFECTION, SEPSIS PROBLEM
3. LATE PHASE - AFTER DISCHARGED - SCAR & CONTRACTURE PROBLEMS 4 ACUTE PHASE MANAGEMENT 5 ETIOLOGY 1. FIRE
2. SCALD
3. CHEMICAL SUBSTANCES
4. ELECTRIC & RADIATION
5. SUNBURN
6. STOVE / GAS EXPLOSION
7. BOMB EXPLOSION 6 7 8 9 10 Burn Capillary permeability and osmotic force change Fluid and protein shift Total blood volume have been lost Burn shock 13 DEPTH ASSESSMENT 1. 1 st DEGREE - EPIDERMIS
2. 2 nd DEGREE - SUPERFICIAL - DEEP
3. 3 rd DEGREE - EXTENSION TO MUSCLE / BONE 14 15 16 17
19 WOUND EXTENT WALLACE RULE OF NINE Head & neck 9% --------> 9% Upper extremities 9% --------> 18% Anterior of the body --------------> 18% Posterior of the body ------------> 18% Lower extremities 18% -------> 36% Genital / perineum -------------> 1 %
24 3. SEVERE - 2 nd DEGREE >25% IN ADULTS - 2 nd DEGREE >20% IN JUVENILES - 3 rd DEGREE >10% - AFFECTED HANDS, FACE, EARS, EYES, FEET, AND GENITAL / PERINEUM - INHALATION INJURY, ELECTRICAL INJURY, OR ASSOCIATED WITH OTHER TRAUMAS
25 I. PRIMARY SURVEY II. SECONDARY SURVEY III.INITIAL CARE OF THE BURN WOUND IV. INITIAL LABORATORY STUDIES V. BURN CENTER REFERRAL 26
I. PRIMARY SURVEY : LIKE ANY OTHER TRAUMA
A. AIRWAY & CERVICAL SPINE PROTECTION B. BREATHING & VENTILATION C. CIRCULATION & HEMORRHAGE CONTROL D. DISABILITY NEUROLOGICAL EXAMINATION E. EXPOSURE
II. SECONDARY SURVEY :
A. HISTORY TAKING B. PHYSICAL EXAMINATION / HEAD TO TOE EXAMINATION C. PRINCIPALS : 1. STOP THE PROCESS CAUSING BURN WOUNDS 2. UNIVERSAL PRECAUTION, HIV, HEPATITIS 3. FLUID RESUSCITATION : 2-4 CC RL X KG BW X %WOUND SURFACE 4. VITAL SIGN 5. NASOGASTRIC TUBE / IF NECESSARY 6. URINARY CATHETER / IF NECESSARY 7. PERFUSSION ASSESSMENT 8. CONTINUED VENTILATORY ASSESSMENT 9. PAIN MANAGEMENT 10.PSYCHOSOCIAL ASSESSMENT 11.TETANUS TOXOID PROFILAXIS 12.MEASURING BODY WEIGHT 13.WOUND CLEANSING (OPERATING THEATRE, GENERAL ANAESTHESIA) 14.ESCHAROTOMY & FASCIOTOMY
29 FLUID RESUSCITATION EVANS FORMULA
BROOKES FORMULA
PARKLANDS FORMULA
BROOKES MODIFICATION
MONAFOS FORMULA
BAXTERS FORMULA DR. SOETOMO GENERAL HOSPITAL
DAY 1 : ADULT: RL 4 CC X KG BW X %WOUND SURFACE / 24 HRS ANAK : RL : DEXTRAN = 17 : 3 2 CC X KG BW X %WOUND SURFACE + MAINTENANCE
MAINTENANCE : < 1 YR : BW X 100 CC 1-3 YRS : BW X 75 CC 3-5 YRS : BW X 50 CC
IN FIRST 8 HRS NEXT 16 HRS
DAY 2 :
ADULT : MAINTENANCE ALBUMIN (IF NECESSARY)
JUVENILE : MAINTENANCE
32 MONITORING FLUID RESUSCITATION 1. URINARY PRODUCTION PER HOUR ADULT : 0,5 CC/BW/HR (30-50 CC/HR) JUVENILE : 1 CC/BW/HR 2. OLIGURIA ASSOCIATED WITH SYSTEMIC VASCULAR RESISTANCE & CARDIAC OUTPUT RECUCTION 3. HAEMOCHROMOGENURIA (RED PIGMENTED URINE) 4. BLOOD PRESSURE 5. HEART RATE 6. HAEMATOCRITE & HAEMOGLOBIN 33 CLOSED WOUND MANAGEMENT WOUND CLEANSING, DEBRIDEMENT, & DESINFECTION WITH SAVLON 1 : 30 TULLE TOPICAL SILVER SULFADIAZINE (SSD) THICK STERILE GAUZE / ELASTIC BANDAGE OPEN THE WOUND DRESSINGS AT DAY 5 UNLESS THERE IS ANY SIGN OF INFECTION PERFORM UNDER GENERAL ANAESTHESIA (IN THE OPERATING THEATRE) III. LABORATORY EXAMINATION
35 Functions of the skin Protection intact skin is the first line of defense against bacterial and foreign-substance invasion Heat regulation Sensory preception Excretion Vitamin D production Expression important with body image - fear of disfigurement
36 STAGES OF BURNS Hypovolemic state begins at the onset of burn and lasts for the first 48 hours - 72 hours Rapid fluid shifts - from the vascular compartments into the interstitial spaces Capillary permeability with burns increases with vasodilation Fluid loss deep in wounds Initially Sodium and H2O Protein loss - hypoproteninemia Hemoconcentration - Hct increases Low blood volume, oliguria Hyponatremia - loss of sodium with fluid Hyperkalemia - damaged cells release K, oliguria Metabolic acidosis
37 STAGES OF BURNS Diuretic Stage begins 48 - 72 hours after burn injury: Capillary membrane integrity returns Edema fluid shifts back into vessels - blood volume increases Increase in renal blood flow - result in diuresis (unless renal damage) Hemodilution - low Hct, decreased potassium as it moves back into the cell or is excreted in urine with the diuresis Fluid overload can occur due to increased intravascular volume Metabolic acidosis - HCO 3 loss in urine, increase in fat metabolism
38 SIGNS OF ADEQUATE FLUID RESUSCITATION : Clear sensorium Pulse < 120 beats per minute Urine output for adults 30 - 50 cc/hour Systolic blood pressure > 100 mm Hg Blood pH within normal range 7.35 - 7.45
39 Organisms that usually infect burns are: a. Staphylococcus aureus
b. Pseudomonas Infection is usually the cause of any deterioration
40 Signs of Sepsis: a. Change in sensorium b. Fever c. Tachyapnea d. Paralytic ileus e. Abdominal distention f. Oliguria
41 Ways to prevent infection:
a. Gowns, masks, gloves
b. Sterile linen
c. Persons with URI should not come in contact with patient
42 WOUND CARE PRINCIPLES 1. GOALS 1. close wound as soon as possible 2. prevent infection 3. reduce scarring and contractures 4. provide for comfort 2. Wound cleaning + closed technique 3. Debridement, mechanical, surgical, enzymatic 4. Topical antibacterial therapy mafenide (sulfonamide) sulfadiazine 5. Biological dressing - Homograft (cadaver skin ) - Heterograft - Autograft 43 IV. BURN CENTER REFERRAL REFERRAL CRITERIA 1. 2nd degree >10% 2. Affecting face, hands, genital, perineum, & main joints 3. 3 rd degree 4. Electric injury 5. Chemical injury 6. Inhalation injury 7. Juveniles 8. Associated with other traumas 44 ACUTE BURN
A. Airway : inhalation injury B. Breathing : fullthickness circumferntial burn C. Circulation : syok Carbon monoxide poisoning Inhalation injury above the glottis Inhalation below the glottis Any victim, burned in a closed area, like a house fire, should be presumed to have an inhalation injury until proven otherwise INHALATION INJURY