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Nurul Najiha binti Noor Azhar 11 2011 141 dr. Johan Lucas, Surgoen Syrgery Department RSUD Ciawi
Identity
Medical history
Physical examination
Pulse rate :
Respiration rate : temperature :
104 x / menit
40 x/ menit 38.8 C
Differential diagnosis
Peritonitis due to rupture of amoebic hepar abcess Peritonitis due to rupture of pyogenis hepar abcess Peritonitis due to perforate appendicitis Empyema Infectious hepatitis
Additional workup
Laboratorium : fecal culture, LFTs X-ray : abdominal erect, abdominal supine, thorax xray. USG / scanning
Investigation (Laboratorium)
Tests Hemoglobin Hematocrit Leukocyte 35% 17 300/uL Result 12,0 mg/dL
569 000/uL B
+ 75 U/L 69 U/L 46 mg/dL 1,26 mg/dL 118 mEq/L 3,9 mEq/L 90 mg/dL 146 mg/dL
Working diagnosis
Management
Non medical treatment :
NGT decompresion Urine catheter Aspiration abcess Pro Exploratory laparotomy
Medication :
IVFD RL 14 gtt/min Metronidazole 3x750 mg drip
Ceftriaxone 1x2 g iv
Ranitidin 3x1 amp iv Ketesse 3x1 amp iv
Prognosis
Thank you
Phase 2 (prodromal phase) Patients experience anorexia, nausea, vomiting, alterations in taste, arthralgias, malaise, fatigue, urticaria, and pruritus, and some develop an aversion to cigarette smoke; when seen by a health care provider during this phase, patients are often diagnosed as having gastroenteritis or a viral syndrome Phase 3 (icteric phase) Patients may note dark urine, followed by pale-colored stools; in addition to the predominant gastrointestinal (GI) symptoms and malaise, patients become icteric and may develop right upper quadrant pain with hepatomegaly
Phase 4 (convalescent phase) Symptoms and icterus resolve, and liver enzymes return to normal