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<discipline>кщр

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/\Which disorders of acid-base balance can cause alveolar hypoventilation?
+ mixed acidosis
- non-gaseous alkalosis
+ respiratory acidosis
- respiratory alkalosis
\/
/\How neuro-muscular excitability change at non-compensated respiratory
alkalosis?
- decrease
+ increase
- not change
\/
/\Which disorders of acid-base balance are accompanied with activation of
acidogenesis and amoniogenesis in kidney?
- renal acidosis
+ metabolic acidosis
- metabolic alkalosis
\/
/\reasons of respiratory alkalosis:
+cerebral tumors, encephalitis with activation of respiratory center
+hysteria
-pneumonia
+mountain sickness
\/
/\Acidity of urine at renal acidosis is decreased:
+correct
-wrong
\/
/\What sentence is correct?
-renal acidosis is developed at activation of acidogenesis and ammoniogenesis
in renal tubules
+ renal acidosis is developed at inhibition of acidogenesis and ammoniogenesis
in renal tubules
\/
/\Respiratory alkalosis is characterized:
+ decreasing P СО2 and decreasing of standard bicarbonate in blood
- decreasing Р СО2 and increasing of standard bicarbonate in blood
- increasing Р СО2 and increasing of standard bicarbonate in blood
\/
/\Metabolic acidosis is characterized:
- decreasing Р СО2 and decreasing of standard bicarbonate in blood
+ secondary decreasing Р СО2 and decreasing of standard bicarbonate in blood
- increasing P СО2 and increasing of standard bicarbonate in blood
\/
/\Respiratory acidosis is characterized:
-increasing P CO2 and decreasing of standard bicarbonate
-decreasing P CO2 and decreasing of standard bicarbonate
+increasing P CO2 and increasing of standard bicarbonate
\/
/\How acidity of urine change at metabolic acidosis?
+increased
-decreased
-not change
\/
/\pH equal 7,25 is observed at:
-compensated alkalosis
-decompensated alkalosis
-compensated acidosis
+decompensated acidosis
\/
/\Choose the reasons of respiratory acidosis:
-excessive injection of acids
+alveolar hypoventilation
-excessive formation of acidic metabolites
-loss of intestinal juice
-insufficient elimination of acidic metabolites with urine
\/
/\Patient has BE = -7 mmol/l. How to characterize this parameter?
+decreased
-normal
-increased
\/
/\Is it correct that the reason of metabolic acidosis is hypoventilation of
lungs?
-yes
+no
\/
/\Is it correct that the reason of respiratory alkalosis is hypoventilation of
lungs?
-yes
+no
\/
/\ Is it correct that the compensation of respiratory acidosis is accompanied
with activation of acidogenesis and ammoniogenesis in kidney?
+yes
-no
\/
/\Which processes take part in compensation of non-gaseous alkalosis?
+hypoventilation of lungs due to suppression of respiratory center
-hypoventilation of lungs
+decreasing of bicarbonate’s reapsorption in kidney
-increasing of bicarbonate’s reapsorption in kidney
-increasing elimination of ammonia chlorine with urine
+decreasing elimination of ammonia chlorine with urine
+decreasing acidity of urine
-increasing acidity of urine
\/
/\Decreased pH is obligatory sign of compensated metabolic acidosis?
-yes
+no
\/
/\Decreased pH is obligatory sign of decompensated metabolic acidosis?
+yes
-no
\/
/\reasons of respiratory acidosis:
-starvation
-nausea
-fistula of stomach
-mountain sickness
+poisoning with barbiturates
+pneumonia
+bronchospasm
\/
/\Can level of glucose into blood characterize of acid-base metabolism at the
patients with diabetes mellitus?
-yes
+no
\/
/\Metabolic acidosis is accompanied with decreasing of standard buffer (SB) in
blood?
+yes
-no
\/
/\Reasons of non-gaseous acidosis:
+diarrhea
-nausea
-emphysema
+oligouria
-excess of steroid hormones
-constipation
-edema of lungs
+starvation
\/
/\Which processes take place at compensation of respiratory alkalosis:
-hyperventilation
+association of kationes with protein buffer
+exit of H+ from bones into blood instead of Са++
-increased reapsorption of bicarbonates in kidney
+elimination of biphosphate Nа with urine
+decreased reapsorption of bicarbonates in kidney
-increased secretion of Н+ into kidney epithelium
+decreased secretion of Н+ into kidney epithelium
\/
/\Reasons of respiratory alkalosis:
-hypoventilation of lungs
+hyperventilation of lungs at irritation of respiratory center
+hyperventilation of lungs at artificial ventilation
-chronic insufficiency of blood circulation
+decreased concentration of oxygen in air
+heat dyspnea
\/
/\Reasons of eliminative acidosis:
+diarrhea
-pneumothorax
-hypoxia
-hyperventilation
-diabetes mellitus
+diseases of kidney with disorders of secretion
\/
/\Which processes support permanent condition of pH in organism:
+reapsorption of bicarbonates
+acidogenesis
-reapsorption of glucose
+ammoniogenesis
-secretion of urea
-production of hemopoetines
-synthesis of rennin
+elimination of organic acids
\/
/\Mechanisms of H+ elimination by kidney:
+elimination of organic acids with urine
-elimination of Н3РО4 with urine
-elimination of Н2SО4 with urine
-reapsorption of bicarbonates
\/
/\Which processes take place at compensation of disorders acid-base metabolism?
–secretion of urea
-reapsorption of glucose
+reapsorption of bicarbonate
+dilution of acids and alkaline in inter-cellular fluid
-synthesis of renin
+activation of protein buffer
\/
/\Limits of рН in blood at compensated disorders of acid-base metabolism?
-7,30 - 7,50
+7,35 - 7,45
-7,30 - 7,35
\/
/\Reasons of non-gaseous alkalosis:
+loss of stomach juice
+constipation
-diarrhea
+excess of steroid hormones
-injuries of liver
-massive inflammation
\/
/\Which processes take place at compensation of respiratory acidosis:
+association of Н+ with hemoglobin
-decreased reapsorption of bicarbonates in kidney
+increased reapsorption of bicarbonates in kidney
-releasing of Н+ from proteins instead of Na+ and К+
+activation of acidogenesis and ammoniogenesis
+elimination of sodium monophosphate with urine
+exchange НСО3 - and Сl - between plasma and red cells
\/

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