Immunity the injured area Supersor T cell Modulate response TYPES OF IMMUNODISORDER

-ability of the body to defend self again *redness, pain, hot of B and T cells
infection Color,volor,dolor Memory Cells Remnants of 1. Hypersensitive (allegry to food,
(insert diagram) Basophilis Releases histamine activated T and B drugs, house dust)
Most cells Histamines and other cells that permit
-exaggerated response to something
chemicals ummeable attack or
Saliva Initiates digestion and response to some the majority of simply ignores
stimulates enzymes pathogens
Came from original cells Attack Chemical 2. Immunodeficiency lack of a
Undifferentiated cells Compliment Promotes bacterial deficiency in the number of immune
system membrane work bacterial cells
What is immunity? for destruction by other cells a. Primary- abnormally low and
Recognize an antigen invader and to mount Attack Cells defective lymphocyte since birth
Natural killer Attack and destroy certain
a defense against it resistance to a particular (congenital immunodefiency
cells tumors and virally infected
disease cells (balloon baby))
Macrophages Phagocytosis of pathogenic b. Secondary- initially health,
Antigen- any substances that stimulates the and cell debris deficient or lacking in normal
production of antibodies specific to it Neutrophilis General phagocytosis esp of healthy immune cells (hiv/aids t
Antibody- immunoglobulin produced by a bacteria cells attacked)
plasma cell is specific for/ bind an antigen Eosinophilis Phagocytosis
Inc number of allegys, eat up
allergens
Types of Immunity
1. General or general defense (B,I,ACl.Ach)
2. Specifc or specific defense (Bc,Tc) SPECIFIC DEFENSE MECHANISM
B cells/ Lympho cells Clonal response to
GENERAL DEFENSE MECHANISM specific antigen
Barriers Plasma cells Antibody factories
Skin Physically prevents invasion for specific antigens
of disease causing agents T cells
Mucus and Traps particles from Helper T cell Stimulate B cell and
ciliated cell entering the respiratory cytotoxicT cells
system Cytotoxic T c Clontal response to
Enzymes Attackes pathogens entering specific membrane
the mouth and digestive bond antigen attach
system to a non cells
Inflammation Increase the blood flow to infected cell
EMERGING DISEASE
Appeared first in a narea for the first time
Existed in an area previously and is rapidly
increasing
Infectious disease increase over the past 20
years and will increase in the future
12% of all human pathogens by newly
species/strains of pathogens
-infection (influenza)
-new population(west nile virus)
-ecologic transformation(lyme disease)
-reemerging infections(resistant TB)
5. Economic developemt (inc antibiotic
treat yield of raired coew leads to
antibiotic resistance)
6. Breakdown and public health
7. Poverty and social inequality
a. TB
8. Bioterrorism
9. Ban and inactive system construction
chlamidia and after requisites

*animals passed to human beings(ecological

transformation)
*hosocemial(hospital acquired) MRSA
emeged in hospitals resistant to antibiotics
*zoonotic (animal reservance indicated the
organism with occasional transmission
human)

1. Microbial Adoptation (genetic drift and

shifts_
2. Changing human susceptibility
(immunocompromised state with
HIV/AIDS)
3. Climate and Weather (diseases with
zoonotic)
4. Change in human demographics and
trade rapid travel enable EID to grow
rapidly