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GH and Contest Prep

During contest preparation total daily insulin release decreases due to


a
decrease in calorie intake. As a result there is a corresponding
decrease in blood
glucose levels that predicts these low insulin periods. As a rule CH
administration
during a period in which a glucometer reading of below 65 exists
results in a
significant increase in fat expenditure.
Many have followed this practice and followed with cardio sessions to
heighten
the effect. Interesting, huh? It has been a wise choice to keep glucose
tabs nearby
incase hypoglycemia sets in. (It would have been really embarrassing
to pass-out on a
tread mill and have it toss a beast to the carpet)
PGF-2/IGF-1: Obviously IGF-1 was both anabolic and anti-catabolic.
However
when paired or stacked with PGF-2, a synergistic response was
realized. The
combination was perfect for symmetry adjustments during pre-contest
diets simply
because both were some-what site-specific in action.
As Frank now realized, different muscles have a unique and different
PTOR,
which means such areas as his arms lose mass more quickly than
others. The PGF-
2/IGF-1 stack significantly increased anabolism in those areas either as
he dieted
down or during the last 3 weeks before his contest.
Thyroid Hormone: T-4 and T-3 thyroid hormones increased the BMR and
the
PTOR while significantly increasing nutrient absorption and utilization.
They also had
a very notable synergistic response with AAS and GH/IGF-1. Since T-4
and T-3
increased BMR and a high protein diet was utilized with anabolic
chemistry, the result
was faster fat loss (also better lean mass retention and growth).
Thermalgenics: Thermalgenics increased the number of calories burned
as
heat expenditure. Clenbuterol and Ephedrine also had anti-catabolic
qualities that
also acted synergistically with GH. Caffeine greatly extended the time
period for fat
burning effectiveness of most thermalgenics.
Anti-Estrogens: Estrogen control was absolutely necessary during
contest
prep. Any increase in estrogen activity could have lead to stubborn fat
deposits (or
even female pattern fat deposits), gyno, and serious water retention.
The reader
knows well the fact that estrogen leads to a negative feed-back loop
that inhibits
HPTA function.
However, since contest prep cycles lasted so long, HPTA regeneration
postcycle
was quite necessary anyway. But why would we have added to the
problem to
begin with? No competitor came in truly hard without estrogen
suppression. FACT!!
Cortisol Suppression: During calorie deficit periods, the body increases
catabolic hormone production. Cortisol, the body's main catabolic
hormone
increases protein based tissue wasting by triggering the release of
amino acids from
muscle cells. The amino acids are stripped of their nitrogen
components and then
utilized as an energy source.
When cortisol suppression was utilized the body increased the use of
fats for
fuel. Total cortisol suppression would have been counter-productive as
there existed
an obvious synergy between cortisol and most contest or growth
chemistries relating
to growth, as well as immune function. Over dosing or prolonged use of
cortisol
suppressing drugs would have potentially resulted in serious negative
feed-back
loops.
DNP: DNP affected the Krebs cycle by making the mitochondria create
heat
instead of ATP (energy). It did so by introducing lots of hydrogen ions
between the
two cellular outer membranes. The Krebs cycle is a series of chemical
reactions that
occur within the mitochondria that are responsible for the breakdown
of nutrient
molecules to form water and carbon dioxide as well as ATP. In short,
DNP uncoupled
the process.
About a 50% + increase in metabolic rate occurred with the use of
5mg/kg of
DNP daily. For most beasts this translated into a fat loss of up to 6-10
LBS in a 7-day
span. Most of this heat was surface heat not internal heat so it was
necessary for
Frank to monitor his body temperature; 101-102 degrees was not
uncommon. Also
Frank slept in an air-conditioned room or had a fan on his face while he
slept.
Though I would never claim DNP was safe, I will say a maximum
dosage of 5
mg/kg daily appeared not to be dangerous for most individuals. At a
dosage of as
little as 8 mg /kg daily, DNP could have been DEADLY!!!
During periods of DNP use, Frank experienced major carb craving
periods.
Usually 25-50 grams of peptide or L-Glutamine resolved this. Arginine
worked well
also, but we had another use for this muscle mass amino acid we will
discuss later.
Another factor that fascinated me about DNP was its ability to prevent
fat gain
during insulin use. It did so by screwing up the shape of the insulin
molecule. This
also meant it was far more difficult to go hypoglycemic when DNP and
insulin were
stacked. Obviously DNP use increased the PTOR which then allowed for
increased
calorie counts and therefore increased nutrient availability at the cell.
I personally believed DNP was very useful but had to be utilized with
serious
caution. Some users simply ingested the 5mg/kg dosage first thing in
the morning.
Others broke it up into 2 evenly divided dosages. I liked DNP's ability to
clear
receptor sites. This was especially effective for androgen receptor
clearing and count
up-regulation.
While utilizing DNP, Frank had to train with higher reps and lower
weight
loads. This was because DNP users have experienced serious muscle
cramps and
tears due to interference with ATP re-synthesis and other factors.
Red Blood Cell Expanders: AAS such as Anadrol-50 were sometimes
utilized as
a method of increasing red blood cells count. This allowed a beast to
appear much
fuller and vascular because there was more blood to expand vascular
tissue (Like
veins and muscle).
There is of course a correlation between red blood cell count and
hemocrit. I
have often seen athletes with a hemocrit of over 52 thus exceeding the
safer
reference range. Instead of using one of the many hemocrit control
drugs it has
always been so much healthier to simply donate blood. The reduction
is immediate
and effective...and far more healthy.
Eprex was a protein that aided in inducing an increase in red blood cell
count.
It too has been used pre-contest to create improved vascularity and a
fuller
musculature. It took about a month to become effective when utilizing
daily dosages
of 1000 iu/d subcutaneously (sc). SC injections were far more effective
when Eprex
was the drug. 60 days total use was a maximum use period. A blood
viscosity
increase too high could have resulted in heart, vascular, and brain
damage.
Eprex was often utilized as a method of increasing vascular support for
new
growth potential. As I have explained prior, new tissue only grows if
there is an
existing supply of vascular and nerve tissue as well as enough
blood/nutrients to
adequately supply it. If there is an existing inability for nerves to
communicate with
the cells or a lack of vascular tissue to supply blood, no growth occurs.
Eprex was
employed by some beasts, but others wisely avoided it.
PGE-1: PGE-1 was a prostaglandin of the same group (but not series) as
PGF-2.
The difference was size created from deep site-injections was mostly
cosmetic and
resulted in a size increase lasting 4-6 hours. When 10-20 mg of PGE-1
was injected
directly into a muscle, swelling occurred within about 10 minutes.
Using arms as an
example, 10-20mg was injected deeply into each bicep/tricep, resulting
in a size
increase of 1-2 " per arm.
This has been utilized as a "last minute fix" for obvious lagging body
parts.
PGE-1 came in 1cc vials, providing either 10 or 20 mg. Nollatil,
Kaverject (5cc
ampules) and generic PGE-1. They are both used in medicine to induce
penile
erections for those who have such problems. I find it interesting that
research has
found that regular use, for this purpose, results in permanent size
gains.
Yes, there too. The issue of layers, not including AAS foundation and
estrogen
control, was a matter of synergistically improving results.

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