Rue argfres fafreen-arer
(Curchos-wet let) Laenec (2¢2&) Testes FT F aR aT
Ger ya ot awe ga UT st Carrhoss at ary feat walt we ez
Sa Ot Ft ABT FT Fibrosis THAT F |
TTT —
ante qaq & der at afar Presi at wa freat gy saat
sfrden aft eoraa sre gafet oa we faetes cer qHq
R waa Ht WM faa aa FH FaUH Gs aL HT Tl Tey ate ag
won ag faeiten gt at Ua wear ate 1x at aa F are at aT ae
ae fox ger fat ore & ate gah era Te aT ae aT Te 2, he
aaa § amt ore gi af Ft ax BF at ata a fede Tat EI
weg wa ae ate dia faeitaa wer aa A mae HC TT AT TH AE
faaias cor OI FS HH aH THT A AIT Heat eT W TT THT
mde qa ae oe fat SAH CATT TK START (Fibrous-Tissue)
at oat & t Sa MHTe Fa Tear, We, Chloroform, Phosphorus
anfe @é det ateft afe art A 8 ot ot & ar at ait
qatar Fate & sera alt ater farzea afte ara F arate VT
at war 2 ar aeitart are Infective hepatitis dla dat &
wa Faq B aged Fe Ae oe FAT SAF MATT TA ST TAT
% 1a Toxic Hepatitis, Massive Necrosis At Acute Cirrhosis of
the Liver at Acute Yellow Atrophy of the Liver wea %, BWA
ABT AT TH HHT FH BAT Sl AAT STAC HTASTART TAT IT
a& aenefa, aaa, afeider (Foetor hepaticus) we-fra = #t
safe marr TEq Baa H are St a Bree gle Sl ara F
Batata atest B seret fast aa Hae at & sas GoraTs B ACT
art 31 ae we arate OT EL TST Ta TR aT oT Tea fae
Rea Aral T wat THT A TAT TEM Wat Fouy ya gt
Bale est Te at aa Ae als aT A vers Te Tak Four
qa ga SST Bee Ke AT IY aT ae Z| eeaT Teo ag
der & fe age arare Baer aa HST a HAT Sea Pewee
wee af (Chronic Cirrhosis aT Necrosis of the Laver) wer et
ga Or ar dt sar Te fear orat 2 1 ae fafeaa ae 2 fe ae OHko arafrn fafircet-aret
were foe a Ae aT met wed 1 ale aTAeT aT aT
Te FHT AAT ALAC SUT VE, aT TaTAAeA F BIT THT FH Lobules
& daha gat at atadtor deste a fas at wt sas Aq (Necrosis)
ait oPear at art & 1
fasta -—
feats zor F ar wad } ast F aa TAR ATTA ws
Portal Spaces ¥ Siggy at afe ge & flex Lobules % fate
frat & AR gt Fat F areata % Fibroblasts set a1 gl w
sare OM asa ae aT fee fire fre arash Be aT J—Hy fae
% Fo Lobule F ae Ae F IAT HA Lobules & feat at AT
gad ak erg FHT Versa gl Ah oNATYG seat
aa A ave SPT WT Nodules ¥ fawat FX Fa Zee VT AT
Multlobular Cunhosis WY #@r oat & | T Nodules at Fo ayS
Lobules ¥ aaa fafa Ga Tl wy de fates maf FATE
va ast at aft afe (Hypeiplasa) F sem ve VAT BT
HIF aa Te Pseudolobule wet F 1 ats Faas F Portal
Smusoids wi tel aaa & asi gat Fat BT TAT Portal Vein & et
qt Hepatic Autery & fawat ft FT Aol at Nodules F ex 7
Central Vein vt adh deft at oe Peart at aes OTB ae-aS aT
aa ay Lobules are Tat AF TA STAT F are Hepatic Veins
aa ort Sate aaa ore See ae aa Fe Fa eT
Rats afern ara F aS TTA (Hyperplasia) H HOT TEs THT ATHTL
% et TT HS HATA St Ta g I ale A Ta Aa FH ale (Rege-
neration) 4 sfeat aa gant se wa Faw erg at gfe et
at aa FHT ATH F Set (Atophied) Bt HAT Ft WA FHT FH Fe
qa et wet gaa wat ae Sinusoids Wad at we %, fax
Portal Ves a gen arava wt gas ont 31 gaat ofa aE
@rat & fF Portal System H wa afry AAT RC HAT SL aeAT TF
arg & agfad a Ft agg B wee Portal Vem at arart
fart (Stenosed gt) amet ¢ 1 way et F Areata F Bile Cana-
houli age-ae at tact @ Ta Bue Fant F gather fate vet Ste,atat & tt Rah
sdifed arrer ar oer sa Uhr a aga HT gat t 1 at, TT ait Bile
ducts ¥ Esch Col Ta Streptococe: & #reor aa et aT F TAT
Cholangitis #1 Stat ¥ TH Lobules ¥ dta-aita FX SETA Fibrosis
Bae oA aH are Ft FST A aT FHT HT BATT ETS BT
Sat ed Bilary crrhosis FEt FI
san —
wee ay HST TAT FaraeaT FH TEs (¥o-ho aT) ae
Ow sat J Tar feast at ae Ge A afire oa qa gs 1 SAAT
area HUA aah aH aaraeT A aT Si eT AAT FH TAK
ft arg ase adt drat | aie Peete MoT F aft ar a BAe
ag Om det & xaled sera F ge OTF aT —
(2) Taq F er Ge F Gs TAF oT feo oT ATF
a ort F aren att g wale Veh A Ae a oat TMT aT
& for oefa wen S ware geo felt ae ye a oA F
ua Tee at tee aa at oy F ae FA TA FS TEC
draat g 1 Uait BY arte F earl aT fata wach FI
(2) 3a ae Te aTATAr ane safe aT forest F ohare ara &
afre Tt wat oat & (wag Git Ft Portal ferereit
% Gt AK Yoo Millmeteis of Water X 200-¥oo AF
ae oat @) aa arrera ane ante & arax efor aT
(Catanh) % warm gt ate wait Sat cae