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The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No.

3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 53



AbstractIn this study a deterministic mathematical model is developed to investigate the spread of malaria in
Nyamira town. The model has seven non linear differential equations which describe the spread of malaria
with three state variables for mosquitoes and four state variables for humans which
are

and . The existence and stability of disease-free and endemic malaria equilibria is
analyzed. Key to the analysis is the definition of the basic reproductive number
0
which was derived by use of
next generation method. The basic reproduction number for Nyamira town is found to be
0
= 0.0806. The
disease- free equilibrium is locally asymptotically stable, if the basic reproduction number is less than one and
the endemic equilibrium exist provided that the basic reproduction number is greater than one. Ordinary
differential equations were used to model malaria where humans and mosquitoes interact and infect each other.
Numerical simulations show that reducing current biting rate of female Anopheles mosquitoes by 5% and
reducing susceptible humans could assist Nyamira town to achieve malaria free status by the year 2030. If, in
addition, the number of days it takes to recover from malaria infection were reduced to ten days malaria free
status could be achieved by the year 2020.
KeywordsAnopheles Mosquitoes; Basic Reproduction Number; Compartmental Model; Deterministic
Mathematical Model and Malaria.

I. INTRODUCTION
ALARIA is a life threatening infectious disease
caused by a parasite called Plasmodium which is
transmitted through the bites of infected female
anopheles mosquitoes. In the human body the parasites
multiply in the liver and they infect red blood cells. There are
four different species causing the human malaria disease:
plasmodium falciparum, plasmodium vivax, plasmodium
ovale and plasmodium malaria [WHO, 2009, 2010]. Malaria
is a parasitic vector borne disease endemic in many parts of
the world [Nita. H. Shah & Jyoti Gupta, 2013]. Our
motivation was to understand how the disease is spread and
the aim of this research is to develop a model for the spread
of malaria in Nyamira town considering that population is not
constant but there are new borns and migrants whereby
previous models considered constant population. At present
at least 300 million people are affected worldwide. Many
infectious diseases including malaria are preventable, yet they
remain endemic in many communities like Abagusii in
Nyamira town due to lack of proper, adequate and timely
control policies [Anderson & May, 1991]. An infectious
disease is that is transmitted from person to another. The
endemicity and prevalence of malaria varies within and
amongst countries due to factors such as resistance of the
malaria parasites to anti malarial drugs. Ironically in
countries where malaria is most prevalent, its prevention and
necessary health precautions are not often a priority nor a
sustained one. Almost all areas of high endemicity lie in
developing countries where inadequate drainage provides
large stagnant water reservoirs which are ideal breeding sites
for Anopheles mosquitoes. There are about 300 500 million
annual cases of malaria worldwide with 1 3 million deaths
(Roll Back Partnership 2005). About 40% of the worlds
populations live in malaria endemic areas. Although the
incidence of malaria has been rising in the last few decades
due to increasing parasite drug resistance and mosquito
insecticide resistance, recently significant resources have
M
*Department of Pure and Applied Mathematics, Jomo Kenyatta University of Agriculture and Technology, Nairobi, KENYA.
E-Mail: osesam2007{at}yahoo{dot}co{dot}uk
**Department of Pure and Applied Mathematics, Jomo Kenyatta University of Agriculture and Technology, Nairobi, KENYA.
E-Mail: jksigey{at}jkuat{dot}ac{dot}ke
***Department of Pure and Applied Mathematics, Jomo Kenyatta University of Agriculture and Technology, Nairobi, KENYA.
E-Mail: jokelo{at}jkuat{dot}ac{dot}ke
****School of Mathematics, University of Nairobi, Nairobi, KENYA. E-Mail: jmkwoyo{at}uonbi{dot}ac{dot}ke
*****Department of Pure and Applied Mathematics, Jomo Kenyatta University of Agriculture and Technology, Nairobi, KENYA.
Samwel Oseko Nyachae*, Johana K. Sigey**, Jeconiah A. Okello***, James M. Okwoyo**** &
D. Theuri*****
A Study for the Spread of Malaria in
Nyamira Town Kenya
The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No. 3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 54
been made available to malaria control programs worldwide
to reduce malaria incidence and prevalence. Comparative
knowledge of the effectiveness and efficacy of different
control strategies is necessary to design useful and cost
effective malaria control programs. The simulations are
conducted using a statistical computing program known as R
[Danso-Addo, 2009].
II. LITERATURE REVIEW
The literature on the mathematical modeling for
communicable diseases is vast and plays a unique role in
comparing the effects of control strategies used individually
or in packages [Sandip Mandal et al., 2011]. Mathematical
modeling has flourished since the days of Ross (1911), who
was the first to model the dynamics of malaria transmission
and Macdonald (1957) who expounded on Ross work,
introducing the theory of super infection. Using data from the
Garki project (1980) many studies have been carried out on
the epidemiology of malaria and one of the most outstanding
is the mathematical model proposed by Dietz et al., (1993)
which Nedelman (1983) analyzed in detail [Gatton et al.,
1996]. Further works on the subject include: Singer et al.,
(1996) and the review by Nedelman. In a Ph D. dissertation,
Chitnis (2008) described a compartmental model for malaria
transmission based on a model by Ngwa & Shu (2000);
defined a reproduction number R
0
for the expected number of
secondary cases that one infected individual would cause
through the duration of the infectious period and showed the
existence and stability of diseases free equilibrium points
and endemic equilibrium points. He computed the sensitivity
indices for R
0
and the parameters in the model. Chitnis et al.,
(2006) presented a similar bifurcation analysis of an
extension to the model in Chitnis (2008), defining R
0
and
showing the existence and stability. This model will extend
the model in Chitnis et al., (2006) and evaluate the sensitivity
indices in R
0
. This model is different from previous models
in that it generalizes the mosquito biting rate and immigration
in a logistic model for the human population with disease
induced mortality. Previous models assumed that mosquitoes
have a fixed number of bites per unit time. This model allows
a more realistic modeling of situations where there is a high
ratio of mosquitoes to humans and where human availability
to mosquitoes is reduced through vector control interventions.
Human migration is common in most parts of the malaria
endemic world and plays an important role in malaria
epidemiology [Diekmann & Heesterbeek, 1999]. As in Ngwa
& Shu (2000) this model also allows humans to be
temporarily immune to the disease, while still transmitting
malaria to mosquitoes. Strategies for controlling the
epidemiology of many infectious diseases such as malaria
include a rapid reduction in both the infected and susceptible
population via treatment as in Tumwiine et al., (2007)
mathematical model for the transmission of malaria. The
dynamics of many epidemic models has been extensively
analyzed in the assumption that the duration of immunity is
independent of exposure Hence the conventional definition of
immunity as absolute refractoriness to infection may be too
restrictive as immunity may confer protection against severe
illness without eliminating chronic, mild infections. Thus
asymptomatic immune carriers may be infective. This
phenomenon of incomplete immunity permitting transmission
is known to exist for malaria and complicates disease control
strategies as the reservoir of infection now includes
symptomatic and asymptomatic infected individuals. It is
clear that the assumption of constant population size in
epidemiological models which is relatively valid when
studying diseases of short duration with limited effects on
mortality may no longer be valid when dealing with endemic
diseases such as malaria [Koella & Antia, 2003]. In such
diseases the effects of changes in population size and disease
induced mortality are far from negligible and in fact can
have a crucial influence on the dynamics of the disease. In a
Masters Dissertation, Mwamtobe (2010) developed a basic
deterministic malaria model with two latent periods in the
non-constant host-vector populations and formulated a model
with intervention strategies by adding the protected and
treated classes in order to assess the potential impact of
protection and treatment strategies on the transmission
dynamics of malaria.
III. MODEL FORMULATION
The human population is divided into compartments:
susceptible humans (

), Exposed humans (

), Infectious
humans (

) and Recovered humans (R).The mosquitoes are


divided into three Compartments: susceptible mosquitoes
(

), Exposed mosquitoes (

) and Infectious mosquitoes


(

). Applying the definitions of the state variables,


parameters interpretation from table 1, 2 and figure 1, the
system of non linear differential equations which describe
the dynamics of malaria are formulated as below:
Table 1: State Variables for the Malaria Model (1)
Parameter Description

Susceptible humans

Exposed humans

Infectious humans
R Recovered humans

Susceptible mosquitoes

Exposed mosquitoes

Infectious mosquitoes
Table 2: Model Parameters and their Interpretations for the Malaria
Model
Parameter Description
Recruitment rate of humans
Recruitment rate of mosquitoes


Force of infection of humans from susceptible state to
exposed state


Force of infection of mosquitoes from susceptible
state to exposed state


Rate of progression of humans from the exposed state
to the infectious state


Rate of progression of mosquitoes from the exposed
state to the infectious state
The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No. 3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 55

Clinical treatment-recovery rate of humans from the
infectious state to the recovered state
Natural death rate for humans

Death of mosquitoes caused by natural death rate and
insecticides
Disease-induced death rate for humans
Rate of loss of immunity for humans


Probability of transmission of infection from an
infectious mosquito to a susceptible
human provided there is a bite


Probability of transmission of infection from an
infectious human to a susceptible
mosquito provided there is a bite
Biting rate of mosquitoes

= +

S
h
S
h

S
h

E
h

E
h
I
h


( + ) I
h

= I
h
R R

S
m


S
m

S
m

E
m
E
m

E
m
I
m

(1)

Figure 1: Compartmental Model for Malaria Transmission
with the initial conditions:
S
h
(0) = S
h0,
E
h
(0) = E
h0
, I
h
(0) = I
h0
, R (0) = R
0
, S
m
(0) =
S
m0
, E
m
(0) = E
m0
, I
m
(0) = I
m0

where

and

equations (1) is the


malaria model.
In the model, the term

denotes the rate at which


the susceptible humans

, becomes infected by infectious


female mosquitoes

and

refers to the rate at which


the susceptible mosquitoes

are infected by infectious


humans

. It was important to note that the rate of infection


of susceptible human

by infected mosquito

is
dependent on the total number of humans

available per
vector,
The total population sizes are N
h
= S
h
+ E
h
+ I
h
+ R

and
N
m
= S
m
+ E
m
+ I
m
with their differential equations


(2)
3.1. Disease Free Equilibrium
Disease free equilibrium points are steady state solutions
where there is no malaria in the human population or
plasmodium parasite in the mosquito population. We can
define the diseased classes as the human or mosquito
populations that are exposed or infectious that is,

and

. In the absence of the disease this implies


that is

= 0 and when the right hand side


of a non linear system (1) is set zero we have
+



= 0

= 0

I
h


( + ) I
h
= 0

I
h
R R = 0

= 0

= 0

= 0
The above equations reduces to

0
= 0

0
= 0
(3)
this implies that

0
=

0
=


(4)
Thus the disease free equilibrium point of malaria
model (1) is given by;
E
0
= (

0
,

0
,

0
,
0
,

0
,

0
,

0
) = (

, 0,
0, 0,

, 0, 0)
(5)
which represent the state in which there is no infection in the
society.(in the absence of malaria)
3.2. Basic Reproduction Number,


We use the next generation operator approach as described by
Diekmann et al., (1990) to define the basic reproduction
number R
0
as the number of secondary infections that one
infectious individual would create over the duration of the
infectious period, provided that everyone else is susceptible.
Reproduction number R
0
is the threshold for many
epidemiology models, it determines whether a disease can
invade a population or not. When R
0
< 1 each infected
individual produces on average less than one new infected
individual so we would expect the disease to die out. On the
other hand if R
0
>1,each individual produces more than one
new infected individual so we would expect the disease to
spread in the population. This means that the threshold
quantity for eradicating the disease is to reduce the value of
R
0
to be less than one. We determine R
0
using the next
generation approach.
The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No. 3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 56
The following steps are followed to compute the basic
reproduction number
0

The basic reproduction number cannot be determined
from the structure of the mathematical model alone but
depends on the definition of infected and uninfected
compartments. Assuming that there are compartments of
which the first compartments to infected individuals.

() =

+
() where

+
() is the rate of
transfer of individuals into compartment by all other means
and

() is the rate of transfer of individual out of the


compartment.
It is assumed that each function is continuously
differentiable at least twice in each variable. The disease
transmission model consists of nonnegative initial conditions
together with the following system of equations:

() =

()

() , = 1,2,3, .
where is the rate of change of .
The next is the computation of the square matrices and
of order , where is the number of infected
classes, defined by =

(
0
) and =

(
0
) with
1 , , such that is nonnegative , is a non singular
matrix and
0
is the disease free equilibrium point (DFE).
Since is nonnegative and nonsingular, then
1
is
nonnegative and also
1
is nonnegative. Hence the matrix
of
1
is called the next generation matrix for the model.
Finally the basic reproduction number
0
is given by

0
= (
1
)
where () denotes the spectral radius of matrix and the
spectral radius is the biggest nonnegative eigenvalue of the
next generation matrix. Rewriting model system (1) starting
with the infected compartments for both
populations;

and then followed by uninfected


classes;

, ,

also from the two populations, then the


model system becomes

+ +


= +


(6)
From the system (6),

and

are defined as:

(7)
and

+ +

(8)
The partial derivatives of (7) with respect to (

) and
the Jacobian matrix of

at the disease free equilibrium


point (5) is:
F =

0
0
0
0

0
0

0

0
0
0
0

0
0
0

(9)
Similarly, the partial derivatives of (8) with respect to
(

) and the Jacobian matrix of

is:
V =

0
0

0
+ +
0
0

0
0


0
0
0

(10)
The inverse of the matrix is given as:

1
=

1
(

+ )

+ (+ + )
0
0

0
1
( + + )
0
0

0
0
1
(

+ )

+ )

0
0
0
1


Now we have to compute F
1
,
F
1
=

0
0
0
0

0
0

0

0
0
0
0

0
0
0

+ + +
0
0

0
1
+ +
0
0

0
0
1

+

0
0
0
1

0
0

+)(+ + )
0

0
( + + )

(+ + )
0

+ )
0
0
0

0
0
0


Let p =

+ )
, q =

, k =

+)(+ + )
and l
=

(+ + )
.
Thus
F
1
=
0
0

0

0
0

0
0
0

0
0
0
(11)
From (11), we can now calculate the eigenvalues to
determine the basic reproduction number
0
by taking the
spectral radius of the matrix F
1
.
Thus it is computed by = 0, we have

0

0

0
0

= 0
=
0 0

0 0 0

0
0 0


The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No. 3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 57
= 0
3
+ = 0

2
= 0

2
= 0 or
2
= 0
= 0 or =
For the four values the dominant eigenvalue of the
matrix F
1
is = +. Therefore the basic reproduction
number R
0
= .
Thus
R
0
=

+)(+ + )(

+ )
(12)
where:

+
is the probability that a human will survive the
exposed state to become infectious.

+
is the probability that a mosquito will survive the
exposed state to become infectious.

+
is the number of humans that one mosquito
infects during its infectious lifetime provided all humans are
susceptible.

+ + +
is the number of mosquitoes that one
human infects during the during the duration of the infectious
period, provided all mosquitoes are susceptible.
The threshold parameter
0
can be defined as the square
roots of the product of numbers of humans one mosquito
infects during its infectious lifetime (
0
) and the number of
mosquitoes one human infects during the duration of the
infectious period (
0
)

provided all humans and mosquitoes
are susceptible. Therefore,

0

=
0

0

=

+)(+ + )

+ )
2

(13)
Thus

0

=

++ +
(14)
and

0
=

+
2
(15)
where

+ +
is the number of latent infectious produced by
a typical infectious individual during the mean infectious
period.

2
is the number of latent infectious produced by a
typical infectious during the mean infectious period.
IV. METHOD OF SOLUTION
We carry out numerical simulations using a fourth order
Runge - Kutta scheme in Matlab. The parameters in the
malaria model (1) were estimated using clinical malaria data
and demographics statistics. Those that were not available
were obtained from literature published by researchers in
malaria endemic regions and countries which have similar
environmental conditions compared to Nyamira town. The
probability of transmission of malaria infection from
infectious humans to susceptible mosquitoes is estimated to
be 0.42 and we also assume that a person who has completely
recovered from malaria will lose her / his malaria acquired
immunity after 3 months based on information received from
medical malaria researchers. The probability of transmission
of infection from an infectious mosquito to a susceptible
human is estimated to be 0.065.
The table below shows the estimated parameters and
their sources for the malaria model (1).
Table 3: Estimated Parameter Value and their Sources for Malaria Model (1)
Symbol Value Source
0.000051 (2010 est) by 2011 CIA World Factbook
0.071 Niger, 2008

0.071 Malaria.com, 2011

0.091 Chitnis, 2008


0.143
Tumwiine et al (2007), Modelling the effect of
treatment and mosquito control on Malaria
transmission.
0.0000043 Daily Nation, Kenya, UNICEF, 2013
0.04 Estimated
0.0000027 World Malaria Report 2010
0.011 Estimated

0.42 Estimated

0.066 Estimated
0.4 Chitnis, 2008
After substituting the above values in table 3 into the
malaria model (1), we have the following system of non
linear differential equations.

= 0.000051+ 0.11 0.0262

S
h
0.000043S
h

= 0.0262

S
h
0.071 E
h
0.071

= 0.071E
h
0.143 I
h

= 0.143 I
h
0.011R

= 0.071 0.168

S
m


0.04 S
m

= 0.168

S
m
0.131 E
m

= 0.091E
m
0.04 I
m
with initial conditions

(0) = 50,000,

(0) = 2,000,

(0) =
9000, R(0) = 8990,

(0)= 65000,

(0)= 2000 and

(0)
=70000. The reproduction number for Nyamira town is less
than one since less people die from malaria and substituting
the parameter values in the reproduction number gives
0
=
0.0806 1 hence malaria can be eliminated from the town.
V. SIMULATION OF RESULTS

Sh

Sh

Sh

Eh

Eh

Ih

) Ih

Ih

R

R

= +

S
h
S
h

S
h

E
h

E
h
I
h


( + ) I
h

= I
h
R R
(1)
The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No. 3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 58
Our malaria model (1) was used for the spread dynamics
of malaria in Nyamira town. The numerical simulations were
done in a population of 60,000 of Nyamira town.
The compartment

contains individuals who were


susceptible to malaria, compartment

contains individuals
who were exposed to malaria, compartment

contains
individuals who were infectious to malaria and compartment
R contains individuals who have recovered from the disease.
A computer software was used to solve the above
ordinary differential equations known as R which generated
the graphs below:

Figure 2: Plot of Susceptible Humans against Time
The above figure is illustrating that the change in the
state variable of the malaria model shows the dynamics with
time of susceptible humans. Susceptible population increases
then remained constant within the three months.

Figure 3: Plot of Exposed Humans against Time
The above figure illustrates the change in the state
variable of malaria model showing the dynamics with time of
exposed humans. Exposed population initially was high but
as the days progress it reduces as some of the population
enters infectious class and others recover.

Figure 4: Plot of Infectious Humans against Time
Illustrates the change in the state variable of the malaria
model showing the dynamics with time of infectious humans.
The infectious population reduced as some recovers and
others die within the three months.

Figure 5: Plot of Recovered Humans with Time
Illustrates the change in the state variable of the malaria
model showing the dynamics with time of recovered humans.
Recovered population initially was high as time goes by
the recovered people reduce.

Figure 6: Plot of the Four State Variables for Humans against Time
This is a phase portrait illustrating the changes in the
four state variables of the malaria model showing the
dynamics with time of susceptible humans, exposed humans,
infectious humans and shows the dynamics of recovered
humans.
VI. CONCLUSION
We presented and analyzed a mathematical model to better
understand the transmission dynamics of malaria in Nyamira
town. The model considered a changing total population that
incorporated recruitment of new individuals into the
susceptible compartment through either birth or immigration.
Mathematically, we modeled malaria as a 7
dimensional system of ordinary differential equations. We
defined the basic reproduction number,
0
, which provides
the expected number of new infections from one infectious
individual over the duration of the infectious period given
that all other members of the population are susceptible. We
proved that if
0
1, the disease cannot persist in the town
and when
0
1 the disease can persist. We have proved that
the disease free equilibrium is locally and asymptotically
stable when
0
< 1 and unstable when
0
> 1. Ongoing
The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No. 3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 59
research and analysis will hopefully help the world get closer
to eliminating the burden of infectious diseases. The trade
off, however, must be made between treating symptoms and
creating a more volatile and resistant to the disease. Care
must be taken when evaluating treatment plans, which is one
of the most useful ways to leverage mathematical models.
VII. RECOMMENDATION
Malaria eradication remains a big challenge to National
Malaria Control Programme in Kenya and in most developing
countries thus there is need to strengthen the control
strategies so that the spread dynamics is reduced. Hence from
the results of this work it recommended that:
People who are ill should quickly seek treatment at
health centres for effective diagnosis and treatment of
the infected in order to avoid the spread of malaria
within the population.
Research institutions should start researching into
genetically modified mosquitoes that would not be
able to transmit malaria. When the probabilities of
transmission of infection from an infectious human to
a susceptible mosquito and vice versa are made
permanently to zero then we can have genetically
modified mosquitoes existing without malaria
infection in the human population.
The Kenya government through the ministry of health
should set aside funds for research in order to come up
with a vaccine such that infants will be vaccinated to
prevent the spread of the malaria within the
population.
Future models to include the effects of the
environment on the spread of malaria. Some
parameters, such as the incubation period in
mosquitoes and mosquito birth rate depend on
seasonal environmental factors such as rainfall,
temperature, and humidity. We can include these
effects by modelling these parameters as periodic
functions of time. This would provide a more accurate
picture of malaria transmission.
ACKNOWLEDGEMENT
I would like to thank the almighty God for his grace, love,
good health and care He has given me in life and especially
through this study. Thank you to my supervisors, Prof J. K
Sigey, Dr. J. Okwoyo, Dr.J. A. Okello and Dr. D. Theuri.
Your dedication, passion for mathematics and your life are
inspirations for me beyond my research. Thanks for your
time, advice, ideas and for giving me the freedom to follow
my interests. I am incredibly fortunate to have such a wealth
of expertise on the theory of the ordinary differential
equations, fluid flow and heat transfer. It is an honor and
pleasure to learn from you. Thanks also to my course - mates
and friends who have spent many hours discussing and
assisting me on many different aspects of my work.
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on ITNs.
[15] WHO Global Malaria Programme (2010), World Malaria
Report 2010.
[16] Sandip Mandal, Ram Rup Sarkar & Somdatta Sinha (2011),
Mathematical Models of Malaria - A Review, Malaria
Journal, Vol. 10, Pp. 202,
http://www.malariajournal.com/content/10/1/202.
[17] P.M.M. Mwamtobe (2010), Modeling the Effects of Multi-
Intervention Campaigns for the Malaria Epidemic in Malawi,
M. Sc. (Mathematical Modeling) Dissertation, University of
Dar es Salaam, Tanzania.
[18] Nita. H. Shah & Jyoti Gupta (2013), SEIR Model and
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Samwel Oseko Nyachae. He was born on
8th March, 1980 in Kisii town, Nyanza
province, Kenya. He holds a Bachelor of
Education (Science) degree in Mathematics
& Physics with first class honours in 2007
from Egerton University, Kenya and is
currently pursuing a Master of Science
degree in Applied Mathematics from Jomo
Kenyatta University of Agriculture and
Technology, Kenya.

The SIJ Transactions on Computer Science Engineering & its Applications (CSEA), Vol. 2, No. 3, May 2014
ISSN: 2321-2381 2014 | Published by The Standard International Journals (The SIJ) 60
He is currently teaching at Enkinda Mixed S. D. A. Secondary
school since 2008 to date near Nyamira town, Kenya. He has much
interest in the study of spread of malaria in different regions.
Prof. Johana Kibet Sigey. Sigey holds a
Bachelor of Science degree in mathematics
and computer science first class honors from
Jomo Kenyatta University of Agriculture and
Technology, Kenya, Master of Science
degree in Applied Mathematics from
Kenyatta University and a PhD in applied
mathematics from Jomo Kenyatta University
of Agriculture and Technology, Kenya.
He is currently the acting director, jkuat, Kisii cbd where he is also
the deputy director. He has been the substantive chairman -
department of pure and applied mathematics jkuat (January 2007
to July- 2012). He holds the rank of senior lecturer, in applied
mathematics pure and applied Mathematics department jkuat since
November 2009 to date. He has published 9 papers on heat transfer
in respected journals.
Teaching experience: 2000 to date- postgraduate programmes:
(jkuat)
Master of science in applied mathematics
Units: complex analysis I and ii, numerical analysis, fluid
mechanics, ordinary differential equations, partial
differentials equations and Riemannian geometry
Supervision of postgraduate students
Doctor of philosophy: thesis (3 completed, 5 ongoing)
Masters of science in applied mathematics: (13 completed, 8
ongoing)
Dr. Okello Jeconia Abonyo. He holds a PhD in Applied
Mathematics from Jomo Kenyatta University of Agriculture and
Technology as well as a Master of science degree in Mathematics
and first class honors in Bachelor of Education, Science; specialized
in Mathematics with option in Physics, both from Kenyatta
University. I have dependable background in Applied Mathematics
in particular fluid dynamics, analyzing the interaction between
velocity field, electric field and magnetic field. Has a hand on
experience in implementation of curriculum at secondary and
university level. I have demonstrated sound leadership skills and
have ability to work on new initiatives as well as facilitating teams
to achieve set objectives. I have good analytical, design and problem
solving skills.
2011-To date Deputy Director, School of Open learning and
Distance e Learning SODeL Examination, Admission &Records
(JKUAT), Senior lecturer Department of Pure and Applied
Mathematic and Assistant Supervisor at Jomo Kenyatta University
of Agriculture and Technology. Work involves teaching research
methods and assisting in supervision of undergraduate and
postgraduate students in the area of applied mathematics. He has
published 10 papers on heat transfer in respected journals.
Supervision of postgraduate students
Doctor of philosophy: thesis (3 completed)
Masters of science in applied mathematics: (8 completed, 8
ongoing)
Dr. Okwoyo James Mariita. James holds a
Bachelor of Education degree in Mathematics
and Physics from Moi University, Kenya,
Master Science degree in Applied
Mathematics from the University of Nairobi
and PhD in applied mathematics from Jomo
Kenyatta University of Agriculture and
Technology, Kenya.
He is currently a lecturer at the University of
Nairobi (November 2011 Present) responsible for carrying out
teaching and research duties. He plays a key role in the
implementation of University research projects and involved in its
publication. He was an assistant lecturer at the University of Nairobi
(January 2009 November 2011). He has published 7 papers on
heat transfer in respected journals.
Supervision of postgraduate students
Masters of science in applied mathematics: (8 completed and
8 ongoing)
Dr. David Theuri. He is the Chairman of
department of Pure and Applied Mathematics
in Jomo Kenyatta university of Agriculture
and Technology.
Supervision of postgraduate students
Masters in applied mathematics : (8
ongoing)

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